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CSF3 — IL8
Text-mined interactions from Literome
Akahane et al., Arzneimittelforschung 1990
:
The enhanced
G-CSF production from NFSA cells may be
due at least to
interleukin-1 released from muroctasin stimulated macrophages
Leizer et al., Blood 1990
:
Cytokine regulation of colony stimulating factor production in cultured human synovial fibroblasts : I.
Induction of GM-CSF and
G-CSF production by
interleukin-1 and tumor necrosis factor
Jiang et al., J Int Med Res 2010
(Alzheimer Disease...) :
Levels of a7 nAChR protein were significantly increased and levels of
interleukin-1ß , tumour necrosis factor-a and nuclear factor-?B ( NF-?B ) protein were significantly decreased in the brain of APP transgenic mice in
response to
G-CSF
Wang et al., Int J Oncol 1995
:
We report that all-trans retinoic acid ( tRA ) synergizes with LPS to
enhance the production of granulocyte colony stimulating factor (
G-CSF ) in PMA treated cells, whereas the production of granulocyte-macrophage CSF,
interleukin 1-beta (IL-1-beta), and tumor necrosis factor-alpha (TNF-alpha) is minimally affected by tRA
Lloyd et al., J Biol Chem 1995
:
Similarly,
125I-IL-8 ligand binding to PMN is
increased by
G-CSF and decreased by LPS treatment ... The stimulatory
effect of
G-CSF on
IL-8R expression is transcriptional as it is inhibited by actinomycin D and is evident in nuclear run-on analyses
Arnold et al., Immunology 1994
(Respiratory Syncytial Virus Infections) :
In addition, IL-3,
G-CSF , IFN-gamma and TGF-beta 2, albeit at high concentrations,
induced a low
IL-8 release from non infected A549 cells
Pajkrt et al., Blood 1997
(Endotoxemia) :
In group 1,
G-CSF significantly
enhanced the release of tumor necrosis factor (TNF),
interleukin-6 (IL-6), IL-8, IL-1 receptor antagonist (IL-1ra), and soluble TNF receptors
Rougier et al., Cytokine 1998
:
G-CSF stimulated and IL-4
inhibited IL-8 production in 10 % FCS