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LTB — TNF

Text-mined interactions from Literome

Zinetti et al., Cytokine 1998 (Sepsis) : Neither the pro-inflammatory cytokines interleukin 2 (IL-2), TNF-alpha nor LPS alone or in combination with interferon gamma (IFN-gamma) had any effect on LT-beta mRNA expression
Amsellem et al., Pulm Pharmacol Ther 1998 : Moreover, the TNF-alpha induced stimulation of LTB4 release that we demonstrated in PMN-HAEC interaction was also inhibited by loratadine
He et al., J Rheumatol 2002 (Osteoarthritis) : The effect of antiinflammatory cytokines rhIL-4, IL-10, and IL-13 on basal and LTB4 dependent stimulation of IL-1beta/TNF-alpha synthesis was studied under titration conditions ... LTB4 stimulated IL-1beta and TNF-alpha synthesis with an EC50 of 190 +/- 35 and 45 +/- 9 nmol/l, respectively ... The antiinflammatory cytokine IL-4 blocked LTB4 dependent stimulation of IL-1beta and TNF-alpha synthesis
Talvani et al., Infect Immun 2002 (Chagas Disease...) : LTB ( 4 ) also induced significant tumor necrosis factor alpha (TNF-alpha) production, and blockade of TNF-alpha suppressed LTB ( 4 ) -induced NO release and parasite killing
Young et al., Br J Pharmacol 2002 (Inflammation) : 7. The data indicate that firm adhesion and transmigration in mouse cremasteric venules stimulated by IL-1beta, but not TNFalpha , is protein synthesis dependent and mediated by endogenous generation of PAF and LTB ( 4 )
Chen et al., Beijing Da Xue Xue Bao 2006 (Arthritis, Rheumatoid) : To investigate quantification of expression of LTB4 inducing IL-1beta and TNF-alpha at mRNA level in synovial membrane cells of rheumatoid arthritis ... Bestatin ( 100 mg/L ) could also remarkably diminish LTB4 induced mRNA expressions of TNF-alpha ( 86 % ) and IL-1beta ( 79 % ) ... LTB4 of synovial membrance cells in rheumatoid arthritis could induce expressions of TNF-alpha and IL-1beta at mRNA level, and their expression at mRNA level had been quantified successfully
Bauldry et al., J Immunol 1991 : We have further examined the effects of TNF-alpha on arachidonic acid ( AA ) release, LTB4 production, and platelet activating factor (PAF) formation in PMN by prelabeling cells with either [ 3H ] AA or [ 3H ] lyso-PAF, priming with human rTNF-alpha, and then stimulating with the chemotactic peptide, FMLP
Zou et al., Mediators Inflamm 1994 : However, neither TNFalpha nor IL-1beta induced a significant LTB ( 4 ) production in SMC alone or AM alone after 24 h of incubation
Domingos et al., Toxicon 2009 : In contrast to the results obtained with the B subunits, incubation of cells with the A subunit of CT (CTA) inhibit TNF-alpha release induced by native CT, native LT, recombinant LTB and LPS
Xu et al., Rheumatol Int 2010 (Arthritis, Rheumatoid) : Exogenous LTB4 remarkably increased the expression of TNFalpha and IL1beta at both the mRNA level and the protein level
Peng et al., Pharm Biol 2011 (Inflammation) : PRP extracts were found to inhibit the production of TNF-a , IL-1ß, and PGE ( 2 ), reduce the expressions of COX-2 at the mRNA and protein levels induced by LPS, and reduced the production of LTB4 induced by A23187
Piotrowska-Tomala et al., Domest Anim Endocrinol 2012 (Mastitis, Bovine) : Tumor necrosis factor , NONOate, and TNF + IL-1a + NONOate increased LTB ( 4 ) production ( P < 0.01 ), whereas LTC ( 4 ) was increased by LPS, TNF, and IL-1a ( P < 0.01 )
Dubois et al., Am Rev Respir Dis 1989 (Asbestosis...) : We next examined the role of LTB4 in mineral-dust induced TNF production
Luedke et al., Agents Actions 1989 : Effect of tumor necrosis factor on granule release and LTB4 production in adherent human polymorphonuclear leukocytes
Goldman et al., Surgery 1993 : Pretreatment of rats by lavage into airways with actinomycin D, 12 ng in 0.1 ml, minimized LTB4 induced TNF synthesis after 1 1/2 and 3 hours ( 38 and 51 pg/ml ), as well as the delayed diapedesis after 4 hours ( 12 PMN/ml ) ( all p < 0.05 )
Steadman et al., Int J Biochem Cell Biol 1996 : DiC8 and TNF alpha in combination, however, synergistically increased LTB4 release ... In contrast, at concentrations above 10-8 M DiC8, whether in the presence or absence of TNF alpha , LTB4 release was inhibited and this was ameliorated by protein kinase C inhibitors
Ferreri et al., Cytokine 1997 : These results demonstrate that PGE2 does not affect LT-beta , IL-4, or IL-3 in Th2 cells, but inhibits TNF mRNA accumulation and production in this T cell subset
Denizot et al., Biochim Biophys Acta 1998 : Bone marrow stromal cells release PGE2 and LTB4 in response to phorbol myristic acetate ( PMA ) ( 1 microM ) and tumor necrosis factor alpha (TNF-alpha) ( 10 ng/ml )