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EGR3 — JUN

Pathways - manually collected, often from reviews:

• NCI Pathway Database Calcineurin-regulated NFAT-dependent transcription in lymphocytes: EGR3 (EGR3) → JUN/FOS/NFAT1-c-4 complex (FOS-JUN-NFATC1_NFATC2_NFATC3) (transcription, inhibits)
  Safford et al., Nat Immunol 2005
  Evidence: mutant phenotype

Text-mined interactions from Literome

Levkovitz et al., J Neurosci 2001 : Furthermore, the Egr inhibitor blocks the ability of MEKK1 [ mitogen activated protein kinase ( MAPK ) kinase kinase 1 ], an upstream kinase capable of stimulating the JNK ( c-Jun N-terminal protein kinase ) -c-Jun pathway, to induce apoptosis and activate c-Jun
Levkovitz et al., J Neurosci 2002 : A dominant negative Egr inhibitor blocks nerve growth factor induced neurite outgrowth by suppressing c-Jun activation : role of an Egr/c-Jun complex
Song et al., Neuroreport 2012 : However, our data show that neither suppressing Egr activity using a dominant negative mutant nor specifically silencing Egr-1 using small interfering RNA inhibited the activation and expression of c-Jun after activity deprivation in cerebellar granule neurons