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IRF6 — TNFSF13B

Text-mined interactions from Literome

Scapini et al., Blood 2005 (Arthritis, Rheumatoid) : We show that CXCL1/GROalpha, CXCL8/IL-8, C5a, immune complexes, tumor necrosis factor-alpha (TNF-alpha), leukotriene B4, N-formyl-methionyl-leucyl-phenylalanine ( fMLP ), and lipopolysaccharide (LPS) , which by themselves do not induce BLyS de novo synthesis, act as potent secretagogues for BLyS, which is mainly stored in Golgi related compartments within G-CSF treated neutrophils, as determined by immunogold electron microscopy
Moon et al., Free Radic Biol Med 2006 : BAFF expression was augmented by LPS stimulation and by serum deprivation that induced ROS production
Alsaleh et al., Arthritis Rheum 2007 (Arthritis, Rheumatoid) : In contrast to IFNgamma, neither TNFalpha, LPS , BLP, nor CpG induced the de novo synthesis and release of BAFF by FLS
Chu et al., J Immunol 2007 : Stimulation of B2 and B1 cells with LPS or CpG-oligodeoxynucleotides induced MyD88 dependent plasma cell differentiation and intracellular expression of BAFF and APRIL
Moon et al., Int Immunopharmacol 2011 (Lupus Erythematosus, Systemic) : Chemicals also inhibited LPS stimulated BAFF production from splenocytes
Moon et al., Exp Cell Res 2011 : Lipopolysaccharide (LPS) increased BAFF expression through the activation of toll-like receptor 4 (TLR4) dependent signal transduction