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IL10 — SYT1

Text-mined interactions from Literome

Du et al., Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi 2004 : NF-kappaB p65 activity in mesangial cells may be inhibited by IL-10 secreted by splenic lymphocytes from the oral immune tolerance rats
Saraiva et al., J Immunol 2005 : In support of a function for this site, NF-kappaB p65/RelA was recruited to HSS-4.5 in vivo and its activation was required for optimal IL-10 gene expression in LPS stimulated macrophages
Zhou et al., J Neurochem 2009 : IL-10 activation of the canonical nuclear factor kappaB pathway enhanced translocation of p50 and p65 and enhanced their binding to kappaB DNA sequences, with p50 playing a more prominent role in neuronal survival
Ember et al., J Gen Virol 2012 (Poxviridae Infections...) : Consistent with this, C4 inhibited interleukin-1ß induced translocation of p65 into the nucleus
Byun et al., Biochem Biophys Res Commun 2012 (Inflammation) : In addition, EGCG treated DCs inhibited lipopolysaccharide (LPS) induced production of pro-inflammatory cytokines ( tumor necrosis factor [TNF ] -a, interleukin [ IL]-1ß, and IL-6 ) and activation of mitogen activated protein kinases ( MAPKs ), e.g., extracellular signal regulated kinase 1/2 ( ERK1/2 ), p38, c-Jun N-terminal kinase (JNK), and nuclear factor ?B ( NF-?B ) p65 translocation through 67LR