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CCK — VIP

Text-mined interactions from Literome

Norlén et al., Br J Pharmacol 2001 : 5. While gastrin, sulfated-cholecystokinin-8 , met-enkephalin and isoprenaline induced a sustained elevation of the submucosal histamine concentration, endothelin, peptide YY, pituitary adenylate cyclase activating peptide, vasoactive intestinal peptide , noradrenaline and adrenaline induced a transient elevation
Katsushima et al., Am J Physiol 1990 : CCK induced downregulation of VIP and secretin receptors was associated with the diminished acinar response to VIP or secretin induced adenosine 3',5'-cyclic monophosphate accumulation and amylase secretion, whereas neither Bt2cGMP nor nitroprusside affected VIP induced amylase secretion
Nakano et al., Regul Pept 1988 : Carbachol ( 10 ( -9 ) to 10 ( -6 ) M ), VIP ( 10 ( -9 ) M ), secretin ( 10 ( -9 ) M ) and glucose ( 11 mM ) did not stimulate CCK secretion, and the addition of atropine ( 10 ( -5 ) M ) to them led to no significant changes
Wiley et al., J Clin Invest 1988 : These studies demonstrate that CCK-8 relaxes the canine sphincter of Oddi via a noncholinergic, nonadrenergic neural pathway involving VIP
Allard et al., Neuropeptides 1986 : Vasoactive intestinal polypeptide (VIP) inhibits potassium induced release of cholecystokinin (CCK) from rat caudato-putamen but not from cerebral cortex ... CCK release elicited by 40 mM potassium from slices of rat caudato-putamen ( cp ) was inhibited by VIP ... VIP inhibition of CCK release appears to be independent of activation of adenylate cyclase because treatment of cp slices with forskolin ( 2 X 10 ( -6 ) to 10 ( -4 ) M ) does not mimic the inhibitory action of VIP
Grider et al., J Nutr 1994 : Based on mimicry by infusion of cholecystokinin to postprandial levels and the ability of specific cholecystokinin receptor antagonists to abolish effects of endogenous cholecystokinin released in response to a meal, three physiological effects of cholecystokinin, all of which are neurally mediated, have been identified : contraction of the gallbladder mediated by cholecystokinin induced release of acetylcholine, relaxation of the sphincter of Oddi mediated by cholecystokinin induced release of vasoactive intestinal peptide and inhibition of gastric emptying mediated by cholecystokinin induced activation of an inhibitory vago-vagal reflex involving vasoactive intestinal peptide induced relaxation of the gastric fundus
Kitagawa et al., Biomed Pept Proteins Nucleic Acids 1995 : The maximally stimulated amylase secretion by PACAP27 was not enhanced by VIP or secretin, but was synergistically increased by cholecystokinin and A23187