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EGFR — TYRP1
Text-mined interactions from Literome
Lammering et al., Int J Radiat Oncol Biol Phys 2001
(Breast Neoplasms...) :
The overexpression of
EGFR-CD533 after infection with Ad-EGFR-CD533 completely
inhibited the radiation induced stimulation of EGFR
Tyr-P relative to the immediate 2.4- to 3.1-fold increases in EGFR Tyr-P in control infected cells ( Ad-LacZ )
Zhang et al., J Pathol 2013
:
The MUC1-CT/p120ctn interaction was highly dependent on
EGFR/Src/Jnk mediated
tyrosine phosphorylation (TyrP) of MUC1-CT. Accordingly, EGFR, Src or Jnk inhibitors ( AG1478, PP2, SP600125, respectively ) abrogated Smk induced MUC1-CT-TyrP, MUC1-CT/p120ctn interaction, AJ disruption, and loss of cellular polarity