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Nakano et al., Eur Surg Res 2000 (Cholestasis) : We investigated whether rats with obstructive jaundice produced with bile duct ligation for 2 weeks are more susceptible to the additional stress of lipopolysaccharide (LPS) administration than sham operated rats and also examined the effects of N-acetylcysteine (NAC) on LPS stimulation in rats with bile duct ligation
Chan et al., Am J Respir Cell Mol Biol 2001 (MAP Kinase Signaling System) : Here we show that in a serum depleted environment ( 0.1 % fetal bovine serum ), NAC substantially inhibited lipopolysaccharide (LPS) activation of the mitogen activated protein kinases ( MAPKs ), namely extracellular signal regulated kinase ( ERK ), p38mapk, and c-Jun NH2-terminal kinase (JNK)
Reis et al., J Neurosci Res 2006 (Neuroblastoma) : The increased H-ferritin mRNA levels in response to LPS are not affected by DPI, NAC , or AG
Paromov et al., BMC cell biology 2008 : Although NAC reduced oxidative stress in LPS stimulated macrophages treated with CEES, it did not reverse CEES induced loss of NO production
Tanaka et al., Biochem Biophys Res Commun 1997 (Astrocytoma) : Transient expression analysis using the chloramphenicol acetyltransferase assay revealed that activation of the IL-8 promoter by LPS was stimulated by BSO and was suppressed by NAC ; likewise LPS induced activation of both NF kappa B and AP-1 was enhanced by BSO and inhibited by NAC
Bellezzo et al., J Lab Clin Med 1998 (Liver Diseases) : NAC and alpha-TOC also markedly suppressed LPS induced tumor necrosis factor-alpha (TNF-alpha) mRNA levels and secretion