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ETS1 — PRL

Text-mined interactions from Literome

Tolón et al., Mol Cell Biol 2000 : Here we show that, in the presence of Ets-1 , VDR stimulates the prolactin promoter in a ligand independent manner, behaving as a constitutive activator
Tentler et al., Endocrine 2003 : Selective repression of rat prolactin gene by stable expression of dominant negative Ets in GH4 pituitary cells
Kanyicska et al., Endocrinology 1991 : Different bacterial toxins capable of modifying specific alpha-subunits of G-proteins were used to characterize the guanine nucleotide binding protein ( G-protein ) dependency of the effects of endothelins ( ETs ) on PRL , LH, and FSH secretion
Calogero et al., J Endocrinol 1994 : Indeed, immunoreactive ET, ET mRNA and ET receptors have been found in the brain and the pituitary gland and ETs stimulate arginine vasopressin, LH, FSH, TSH and gonadotrophin releasing hormone and inhibit prolactin release in vitro
Bradford et al., Mol Cell Biol 1997 : These data show that a functional interaction of GHF-1 and Ets-1 , acting via a composite DNA element, is required to establish lactotroph-specific PRL gene expression, thus providing a molecular mechanism by which GHF-1 can discriminate between the GH and PRL genes