Gene interactions and pathways from curated databases and text-mining

◀ Back to IL1A

CD46 — IL1A

Text-mined interactions from Literome

Wuyts et al., Eur Respir J 2003 (Asthma...) : Therefore the current study investigated whether N-acetylcysteine (NAC), an antioxidative agent, inhibits the interleukin (IL)-1beta induced expression and production of eotaxin and monocyte chemotactic protein (MCP)-1 in human airway smooth muscle cells ( HASMC )
Colotta et al., J Immunol 1992 : IL-1 , TNF, IFN-gamma, granulocyte-macrophage-CSF and, to a lesser extent, macrophage-CSF, as well as inactivated streptococci, also induced MCP gene expression
Wang et al., Arterioscler Thromb 1991 : Tumor necrosis factor and bacterial lipopolysaccharide but not IL-6 also induced MCP and IL-8 gene expression in SMCs. Nuclear runoff analysis revealed that IL-1 augmented transcription of the MCP and IL-8 genes
Mahtout et al., FEMS Immunol Med Microbiol 2011 : Although these two cytokines had no effect on CD46 and CD55 gene expression in epithelial cells, IL-1ß and tumor necrosis factor-a induced a significant upregulation
Turner-Brannen et al., Arthritis Res Ther 2011 (Inflammation) : This study demonstrated that IDR-1002 suppressed the production of IL-1ß induced MMP-3 and monocyte chemotactic protein-1 (MCP-1) ; in contrast, IDR-1002 enhanced the production of IL-1RA, without neutralizing all chemokine responses
Honda et al., Anticancer Res 2011 (Colonic Neoplasms...) : RESULTS : In THP-1 cells, monocyte chemotactic protein (MCP)-1 mRNA expression increased in a time dependent manner from day 3 after co-culture with DLD-1 cells ; furthermore, expression of vascular endothelial growth factor ( VEGF ) -A, tumor necrosis factor (TNF)-a, interleukin (IL)-1ß , and IL-8 mRNA was increased from day 5
Paik et al., Molecular vision 2012 (Graves Ophthalmopathy...) : The orbital fibroblasts from patients with TAO were Thy-1- positive fibroblasts ( > 90 % ) with the ability to secrete IL-6, IL-8, monocyte chemotactic protein-1 (MCP-1) , and HA in response to IL-1ß
Timper et al., Am J Physiol Endocrinol Metab 2013 (Insulin Resistance) : GIP induced mRNA expression of IL-6, IL-1ß , and the IL-1 receptor antagonist IL-1Ra, whereas TNFa, IL-8, and monocyte chemotactic protein (MCP)-1 remained unchanged
Huang et al., J Immunol 1998 (Peritonitis) : Mouse MCP-6 did not induce cultured human endothelial cells to express TNF-alpha, RANTES, IL-1alpha , or IL-6