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GRAP2 — TLR5
Text-mined interactions from Literome
Ivison et al., J Immunol 2007
:
Together, these results suggest that phosphorylation of
TLR5 by PKD may be one of the proximal elements in the cellular response to flagellin, and that this event
contributes to
p38 MAPK activation and production of inflammatory cytokines in epithelial cells
Beaudoin et al., Biochem Biophys Res Commun 2012
(Cystic Fibrosis) :
Inactivation of mucA ( the gene responsible for the mucoid phenotype ) in PAO1 leads to
p38a MAPK
activation by both TLR2 and
TLR5 , as observed in the clinical mucoid isolate PACF508