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MYLIP — PIK3R1
Text-mined interactions from Literome
Huang et al., Am J Pathol 2012
(Lymphoma, Large B-Cell, Diffuse) :
Taken together, our results reveal a novel target involved in miR-155 biological characteristics and provide a molecular link between the overexpression of
miR-155 and the
activation of
PI3K-AKT in DLBCL
Song et al., Arch Biochem Biophys 2012
(Neointima) :
Specifically, overexpression of
miR-223 and miR-153 inhibited stretch stress
enhanced VSMC proliferation and the activity of
PI3K-AKT signaling
Yu et al., Cell death & disease 2013
(Cell Transformation, Neoplastic...) :
Our studies are the first to demonstrate that reduced CTGF as an unfavorable prognosis factor mediates the
activation of
miR-18b , an oncomir directly suppresses CTGF expression, by
PI3K/AKT/C-Jun and C-Myc and promotes cell growth of NPC
Yang et al., Biochim Biophys Acta 2013
(Cardiomegaly) :
And
PI3K inhibitor, LY294002, effectively
prevented LPA induced
miR-23a expression in cardiomyocytes, suggesting that LPA might induce miR-23a elevation by activating LPA3 and PI3K/AKT pathway