◀ Back to CCND1
CCND1 — HDAC3
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
-
IRef Biogrid Interaction:
HDAC3
—
CCND1
(physical association, affinity chromatography technology)
Petre-Draviam et al., Oncogene 2005*
-
IRef Biogrid Interaction:
HDAC3
—
CCND1
(physical association, affinity chromatography technology)
Lin et al., J Biol Chem 2002*
-
IRef Biogrid Interaction:
HDAC3
—
CCND1
(physical association, affinity chromatography technology)
Fu et al., J Biol Chem 2005*
-
IRef Biogrid Interaction:
HDAC3
—
CCND1
(colocalization, imaging technique)
Fu et al., J Biol Chem 2005*
-
IRef Biogrid Interaction:
HDAC3
—
CCND1
(physical association, affinity chromatography technology)
Batra et al., Mol Cancer Ther 2010*
-
IRef Hprd Interaction:
Complex of 17 proteins
(in vivo)
Liu et al., Biochem J 2006
-
IRef Hprd Interaction:
HDAC3
—
CCND1
(in vivo)
Lin et al., J Biol Chem 2002*
Text-mined interactions from Literome
Peterson et al., Mol Cell Biol 2007
(Breast Neoplasms) :
SMRT is
required for full expression of the ERalpha target genes
cyclin D1 , BCL-2, and progesterone receptor but not pS2, and its depletion significantly attenuated estrogen dependent proliferation of MCF-7 cells
Song et al., J Biol Chem 2008
:
Overexpression of SMRT and
NCoR attenuated the transcription of beta-catenin-TCF4-specific reporter gene and of
CCND1 , an endogenous beta-catenin target gene