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IL18 — PTBP2
Text-mined interactions from Literome
Manome et al., Immunology 1999
(Dermatitis, Contact) :
Finally, we examined the effects of these chemicals on CD86 expression by three different macrophage subsets and
DCs induced from the cultures of human peripheral blood monocytes in the
presence of macrophage colony stimulating factor ( M-CSF ), M-CSF
+ interleukin-4 (IL-4), granulocyte-macrophage colony stimulating factor ( GM-CSF ), and GM-CSF + IL-4, respectively
Liu et al., Chin J Cancer 2003
(Breast Neoplasms) :
DCs were
stimulated by granulocyte/macrophage colony stimulating factor ( GM-CSF ),
interleukin-4(IL-4) , and tumor antigen
Pietilä et al., J Leukoc Biol 2005
(Salmonella Infections) :
CC chemokine ligand 5 (CCL5), CXC chemokine ligand 10, tumor necrosis factor alpha, interleukin (IL)-12, and
IL-18 gene expression and protein production were readily
induced by Salmonella infected Mphis and
DCs
Tada et al., Infect Immun 2005
:
The stimulation of
DCs with MDP and FK565 in combination with lipid A, poly ( I:C ), and CpG DNA, but not with Pam3CSSNA, synergistically
induced interleukin-12 (IL-12) p70 and gamma interferon ( IFN-gamma ), but not
IL-18 , in culture supernatants and induced IL-15 on the cell surface
Hua et al., Nan Fang Yi Ke Da Xue Xue Bao 2006
:
The
DCs were derived from healthy human peripheral blood monocytes in the
presence of granulocyte-macrophage colony stimulating factor,
interleukin (IL)-4 and tumor necrosis factor (TNF) alpha
Ito et al., Int Immunol 2006
:
IL-18 induced CD11b ( + )
DCs from CD4 ( - ) CD8 ( - ) thymocytes in vitro, which exhibited strong antigen presenting activity and formed conjugates with CD4 ( + ) CD8 ( + ) T cells efficiently
Byun et al., Biochem Biophys Res Commun 2012
(Inflammation) :
In addition, EGCG treated
DCs inhibited lipopolysaccharide (LPS) induced production of pro-inflammatory cytokines ( tumor necrosis factor [TNF ] -a,
interleukin [ IL]-1ß, and IL-6 ) and activation of mitogen activated protein kinases ( MAPKs ), e.g., extracellular signal regulated kinase 1/2 ( ERK1/2 ), p38, c-Jun N-terminal kinase (JNK), and nuclear factor ?B ( NF-?B ) p65 translocation through 67LR
Khanna et al., Transplantation 1998
:
In addition, however, a link was observed between the beneficial effect of donor BM and comparatively large numbers of donor major histocompatibility complex class II ( IAb+ ) -positive cells in recipients ' spleens, and in cultures of granulocyte-macrophage colony stimulating factor
+ interleukin-4 stimulated
DCs from recipients ' BM