◀ Back to PIK3R1
PIK3R1 — TCF19
Text-mined interactions from Literome
Smith et al., J Biol Chem 2005
:
Furthermore, DEX mediated inhibition of
LEF/TCF transcriptional activity was attenuated in the
presence of dominant negative forms of either
PI3K or protein kinase B/Akt
Belaiba et al., Mol Biol Cell 2007
(Anoxia) :
Furthermore, inhibition of the phosphatidylinositol 3-kinase (PI3K)/AKT but not extracellular signal regulated kinase 1/2 pathway blocked the hypoxia dependent induction of HIF-1alpha mRNA and HIF-1alpha promoter activity, suggesting involvement of a
PI3K/AKT regulated
transcription factor
Kim et al., J Immunol 2007
(Asthma...) :
Our results also showed that
PI3K inhibitors or AdPTEN
down-regulated a
transcription factor , NF-kappaB activity, and BAY 11-7085 substantially reduced the increased levels of IL-17 after TDI inhalation
Banerjee et al., Endocrinology 2009
:
Downstream of ERK1/2, CG activates the nuclear
transcription factor , Elk1, also in a
PI3K-MAPK dependent manner
Sonderegger et al., Endocrinology 2010
:
Chemical inhibition of
PI3K abolished Wnt dependent phosphorylation of AKT and GSK-3beta and trophoblast motility but did not
affect appearance of activated beta-catenin or
Wnt/TCF reporter activity
Yen et al., J Biol Chem 2011
(MAP Kinase Signaling System) :
PKA,
PI3K , and ERK inhibitors abolished PGE2- and cAMP induced c-Fos and MMP-9 up-regulation, and ERK activation was
required for the binding of activator protein 1 (AP-1)
transcription factor to the MMP-9 promoter