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AR — EP300

Pathways - manually collected, often from reviews:

• OpenBEL Selventa BEL large corpus: EP300 → AR (directlyIncreases, AR Activity)
  Evidence: In this study, we describe a novel ligand-dependent transactivation function for AR that is independent of its DNA binding ability. AR dramatically increased the intrinsic transcriptional activity of the nuclear receptor coactivators glucocorticoid receptor-interacting protein-1 (GRIP1), cAMP response element-binding protein-binding protein, and p300 that are tethered to DNA.

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Biogrid Interaction: EP300 — AR (physical association, affinity chromatography technology) Dai et al., Carcinogenesis 2008*
• IRef Hprd Interaction: AR — EP300 (in vitro) Fu et al., J Biol Chem 2000*
• IRef Innatedb Interaction: AR — EP300 (unknown, -) Mukhopadhyay et al., Exp Cell Res 2006