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GPI — MET
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Funasaka et al., Cancer Res 2007
(Fibrosarcoma...) :
This study identifies that
MET in HT1080 human lung fibrosarcoma cells was
initiated by down-regulation of the housekeeping gene product/cytokine
PGI/AMF , and the results depicted here suggest a novel therapeutic target/modality for mesenchymal cancers
Funasaka et al., Cancer Res 2009
(Breast Neoplasms) :
Taken together, the results show the involvement of PGI/AMF in both EMT and MET : overexpression of
PGI/AMF induces EMT in normal breast epithelial cells and reduction of PGI/AMF expression
led to
MET in aggressive breast cancer cells
Niinaka et al., Cancer Res 2010
(Lung Neoplasms...) :
Silencing of
AMF/PGI induced
MET , in which upregulation of E-cadherin and cytokeratins, as well as downregulation of vimentin, were noted
Przygodzki et al., J Physiol Biochem 2012
:
Our present study was aimed at verifying whether the increased production of
PGI ( 2 ), a vasodilating prostanoid, in
response to MNA, its metabolite
N-methyl-2-pyridone-5-carboxamide (Met2PY) , and nicotinamide may be reproduced under in vitro conditions
Fantone et al., Biochim Biophys Acta 1984
:
In this study we have examined the
effects of PGEs and
PGI2 on
f-Met-Leu-Phe- and phorbol-myristate-acetate induced rat neutrophil membrane potential changes using an optical probe of membrane potential 3,3-dipropylthiodicarbocyanine iodide