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GRAP2 — TLR5

Text-mined interactions from Literome

Ivison et al., J Immunol 2007 : Together, these results suggest that phosphorylation of TLR5 by PKD may be one of the proximal elements in the cellular response to flagellin, and that this event contributes to p38 MAPK activation and production of inflammatory cytokines in epithelial cells
Beaudoin et al., Biochem Biophys Res Commun 2012 (Cystic Fibrosis) : Inactivation of mucA ( the gene responsible for the mucoid phenotype ) in PAO1 leads to p38a MAPK activation by both TLR2 and TLR5 , as observed in the clinical mucoid isolate PACF508