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CCND3 — MYLIP

Text-mined interactions from Literome

Lu et al., Cancer Res 2011 (Nasopharyngeal Neoplasms) : Mechanistic investigations revealed that miR-26a suppressed the expression of c-myc, the cyclin D3 and E2, and the cyclin dependent kinase CDK4 and CDK6 while enhancing the expression of CDK inhibitors p14(ARF) and p21 ( CIP1 ) in an EZH2 dependent manner
Yu et al., BMC cancer 2012 (Breast Neoplasms) : MiRNA transfection and luciferase assay confirmed that bcl-2 was regulated by miR-16 and miR-143, cyclinD1 was modulated by miR-16
Wang et al., Carcinogenesis 2012 (Carcinoma, Hepatocellular...) : We conclude that the frequently downregulated miR-138 can regulate CCND3 and function as a tumor suppressor in HCC
Li et al., Blood 2013 (Leukemia, Myeloid, Acute) : Suppression of miR-193a expands the oncogenic activity of the fusion protein AML-ETO, because miR-193a represses the expression of multiple target genes, such as AML1/ETO, DNMT3a, HDAC3, KIT, CCND1 , and MDM2 directly, and increases PTEN indirectly
Poudel et al., Biochem Biophys Res Commun 2013 (Neoplasms) : The transient transfection of pre-miR-30C resulted in greater than 70 % growth inhibition in PC-3 cells and provided strong evidence that miR-30C selectively suppresses the expression of cyclin D1 and D2, but not cyclin D3
Furuta et al., PloS one 2013 (Carcinoma, Hepatocellular...) : Among the candidates, we successfully identified CCNE1, CDC25A, CCND3 , CDK4, and BTRC as direct targets for miR-497 and miR-195
Guo et al., Oncol Rep 2013 : Furthermore, the overexpression of miR-708 reduced the expression of Akt1, CCND1 , MMP2, EZH2, Parp-1 and Bcl2 in A172 and T98G cells