◀ Back to JUN

JUN — STK24

Text-mined interactions from Literome

Pacurari et al., Environ Health Perspect 2008 (Neoplasms, Mesothelial) : In the present study, we exposed mesothelial cells to SWCNTs and investigated reactive oxygen species ( ROS ) generation, cell viability, DNA damage, histone H2AX phosphorylation, activation of poly(ADP-ribose) polymerase 1 ( PARP-1 ), stimulation of extracellular signal regulated kinase ( ERKs ), Jun N-terminal kinases (JNKs) , protein p38, and activation of activator protein-1 (AP-1), nuclear factor kappaB (NF-kappaB), and protein serine-threonine kinase ( Akt )
Schinkmann et al., J Biol Chem 1997 : Overexpression of mst-3 wild-type or kinase dead protein affects neither the extracellular signal regulated kinases ( ERK1/2 or ERK6 ), c-Jun N-terminal kinase (JNK) , p38, nor pp70S6 kinase, suggesting that mst-3 is part of a novel signaling pathway