We have a suspicion that you are an automated web bot software, not a real user. To keep our site fast for other users, we have slowed down this page. The slowdown will gradually disappear. If you think this is a mistake, please contact us at genome-www@soe.ucsc.edu. Also note that all data for hgGeneGraph can be obtained through our public MySQL server and all our software source code is available and can be installed locally onto your own computer. If you are unsure how to use these resources, do not hesitate to contact us.
UCSC Genome Browser Gene Interaction Graph
Gene interactions and pathways from curated databases and text-mining

◀ Back to HNRNPF

CCR2 — HNRNPF

Text-mined interactions from Literome

Qu et al., J Exp Med 2004 : However, absence of CCR2 led to an increased labeling of the minor Gr-1int monocyte population, and the number of latex+ DCs that emigrated to LNs was correspondingly increased
Salazar-Gonzalez et al., Immunity 2006 (Salmonella Infections) : Thus, CCR6 dependent regulation of DCs is responsible for localized T cell dependent defense against entero-invasive pathogens
Robays et al., J Immunol 2007 (Asthma) : Using this approach, we show that CCR2 , but not CCR5 or CCR6, directly controls the accumulation of DCs into allergic lungs
Ibarra et al., Immunobiology 2011 (Arthritis, Experimental...) : To examine the mechanism by which DCs contribute to the increased susceptibility of arthritis in Ccr2?/? mice, we tested the hypothesis that CD8a? DCs are protective ( tolerogenic ) against autoimmune arthritis by examining the role of CD8a? DCs in Ccr2?/? and SKG mice
Qiu et al., J Immunol 2012 (Cryptococcosis...) : TLR9 deletion diminished 1 ) accumulation and activation of CD11b ( + ) dendritic cells (DCs) , 2 ) the induction of IFN-? and CCR2 chemokines CCL7, CCL12, but not CCL2, at week 1, and 3 ) pulmonary accumulation and activation of the major effector cells CD4 ( + ) and CD8 ( + ) T cells, CD11b ( + ) lung DCs, and exudate macrophages at week 3