◀ Back to MYD88
MAPK3 — MYD88
Text-mined interactions from Literome
Coste et al., J Clin Invest 2010
(Cell Transformation, Neoplastic...) :
Here we have shown that
MyD88 is also
required in a cell-autonomous fashion for RAS mediated carcinogenesis in mice in vivo and for
MAPK activation and transformation in vitro
Gorina et al., Glia 2011
:
Astrocyte TLR4 activation induces a proinflammatory environment through the interplay between
MyD88 dependent NF?B signaling,
MAPK , and Jak1/Stat1 pathways
Oliveira et al., Infect Immun 2011
(Brucellosis...) :
Extracellular signal regulated kinase 1 ( ERK1 ) and ERK2 and p38 as well as p65 NF-?B phosphorylation was profoundly
impaired in IRAK-4 ( -/- ) and
MyD88 ( -/- ) macrophages activated by Brucella
Dauphinee et al., Am J Physiol Heart Circ Physiol 2011
:
We find that Ga ( i/o ) -mediated activation of the
MAPK is
independent of the canonical
MyD88 , interleukin-1 receptor associated kinase, and tumor necrosis factor receptor associated factor 6 signaling cascade in LPS stimulated cells
Piao et al., J Immunol 2013
(Inflammation) :
Peptides derived from TRAM TIR BB loop ( TM4 ) and C helix ( TM6 ) inhibited the LPS induced activation of
MyD88 dependent and TRIF dependent cytokines, as well as
MAPK activation