◀ Back to FAS

BCR — FAS

Text-mined interactions from Literome

Yoshida et al., Int Immunol 2000 (Immunologic Deficiency Syndromes) : These results indicate that BCR ligation activates an apoptotic signaling pathway distinct from Fas mediated apoptosis in WEHI-231 cells, and that BCR mediated apoptosis of WEHI-231 cells does not require Fas or related molecules such as DR3, DR4 and DR5, as all of these death receptors require FADD/MORT1 and/or CrmA-sensitive caspases for induction of apoptosis
Huang et al., J Biol Chem 2013 (Leukemia, Myelogenous, Chronic, BCR-ABL Positive) : We found previously that expression of the CML related Bcr-abl oncoprotein in myeloid progenitor cells increases expression of Fas associated phosphatase 1 (Fap1)
Galibert et al., J Exp Med 1996 : CD40 + BCR stimulation of resting B cells with CD40 ligand transfected fibroblastic cells in the presence of cross linked anti-BCR monoclonal antibodies resulted in the coexpression of CD38, CD95 , and CPM
Wang et al., J Exp Med 1996 : Moreover, BCR cross linking in Lyn-/- B cells suppressed Fas expression induced by costimulation with CD40 ligand and IL-4
Selleri et al., Blood 1998 (Leukemia, Myelogenous, Chronic, BCR-ABL Positive...) : Fas mediated modulation of Bcr/Abl in chronic myelogenous leukemia results in differential effects on apoptosis