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ICAM1 — ICAM3
Text-mined interactions from Literome
Zhou et al., Mech Ageing Dev 2006
:
In this report,
ICAM-1 accumulates in late passage endothelial cells when compared to early passage endothelial cells, yet
ICAM-1 protein expression is
attenuated when senescent cells are challenged by TNF-alpha ( 10ng/ml )
Tiisala et al., Scand J Immunol 1994
:
In this study the authors investigated whether a quantitative increase in the surface expression of
ICAM-ligands also can
lead to enhanced binding to purified
ICAM-1
Bryant et al., Infect Immun 1996
:
PLC strongly
induced expression of ELAM-1,
ICAM-1 , and IL-8, while PFO stimulated early
ICAM-1 expression but did not promote ELAM-1 expression or IL-8 synthesis
Shen et al., J Virol 1997
:
TNF-alpha significantly
enhanced WNV induced increases in E-selectin, P-selectin,
ICAM-1 , and VCAM-1 expression, while IFN-gamma enhanced WNV induced
ICAM-1 expression
Braun et al., Arterioscler Thromb Vasc Biol 1997
(Second Messenger Systems) :
In cultured human smooth muscle cells from coronary arteries and saphenous veins, tumor necrosis factor-alpha (TNF-alpha) and interleukin-1 beta (IL-1 beta)
induced the expression of
intercellular adhesion molecule ( ICAM-1 ) and vascular cell adhesion molecule ( VCAM-1 ), whereas interferon-gamma (INF-gamma) selectively stimulated the expression of
ICAM-1
Miyatake et al., Nephron 1998
(Disease Models, Animal) :
Although the expression of
ICAM-1 can be induced by the stimulation of inflammatory cytokine,
ICAM-1 expression can also be
induced by such nonimmune mechanisms as shear stress