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UCSC Genome Browser Gene Interaction Graph
Gene interactions and pathways from curated databases and text-mining

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BAX — IGF1

Text-mined interactions from Literome

Chen et al., Life Sci 2000 : IGF I partially suppressed Bax induction, Caspase 3 activation, DNA fragmentation, and increased cardiomyocyte survival
Yamamura et al., Am J Physiol Heart Circ Physiol 2001 (Myocardial Reperfusion Injury) : IGF-I differentially regulates Bcl-xL and Bax and confers myocardial protection in the rat heart ... These results suggest that IGF-1 differentially regulates Bcl-xL and Bax in heart mitochondria, which may be causally related to myocardial protection against ischemia-reperfusion injury
Nakao et al., J Thorac Cardiovasc Surg 2001 (Disease Models, Animal...) : Immunohistochemical studies revealed that insulin-like growth factor 1 increased expression of the antiapoptotic Bcl-xL protein and inhibited expression of the proapoptotic Bax protein in motor neuron cells 24 and 48 hours after the operation
Ness et al., Glia 2004 : Rather, IGF-I prevented the glutamate induced translocation of Bax to the mitochondria ... Rather, IGF-I prevented the glutamate induced translocation of Bax to the mitochondria
Zhou et al., Zhong Xi Yi Jie He Xue Bao 2005 : Immunohistochemical and integral optical density analytic methods were used to observe the effects of Yiqi Huayu Recipe-, Yiqi Recipe-, Huayu Recipe medicated sera and insulin-like growth factor 1 (IGF-1) on the expressions of bcl-2, Bax and caspase-8 of apoptotic anulus fibrosus cells in rats induced by anti-Fas antibody
Kim et al., Mol Reprod Dev 2006 : In both IVF and SCNT embryos, anti-IGF-IR antibody along with IGF-I neutralized the effect of IGF-I on expression of Bcl-2 and Bax genes
Wang et al., Endocrinology 1998 : To investigate IGF-I actions on apoptosis of H9C2 cardiac muscle cells, we have defined the effects of IGF-I on Bcl-2, Bax , caspase 3, DNA fragmentation, and cell survival ... IGF-I partially inhibited Bax induction, caspase 3 activation, DNA fragmentation, and enhanced cell survival