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CDH1 — RBBP7

Text-mined interactions from Literome

Peinado et al., Mol Cell Biol 2004 : Here we show that mammalian Snail requires histone deacetylase (HDAC) activity to repress E-cadherin promoter and that treatment with trichostatin A (TSA) is sufficient to block the repressor effect of Snail
Witta et al., Cancer Res 2006 (Carcinoma, Non-Small-Cell Lung...) : The zinc finger transcriptional repressor, ZEB1, inhibits E-cadherin expression by recruiting histone deacetylases (HDAC)
Villar et al., Infect Immun 2007 (Candidiasis, Oral) : Mucosal tissue invasion by Candida albicans is associated with E-cadherin degradation, mediated by transcription factor Rim101p and protease Sap5p
Iacopino et al., Int J Oncol 2008 (Prostatic Neoplasms) : The goal of this study was to assess the effects of an HDAC inhibitor, valproic acid ( VA ), on proliferation, androgen-sensitivity, androgen receptor levels and E-cadherin ( E-cad ) expression in human prostate cancer cells
Thomas et al., Bioorg Med Chem 2008 (Lung Neoplasms) : Prodrug incubation with beta-glucuronidase in the culture media led efficiently to the release of the parent drug and thereby restoring its ability to decrease cell proliferation, to inhibit HDAC and to induce E-Cadherin expression
von Burstin et al., Gastroenterology 2009 (Lung Neoplasms...) : In line, mesenchymal pancreatic cancer specimens and primary cell lines from genetically engineered Kras ( G12D ) mice showed HDAC dependent down-regulation of E-cadherin and high metastatic potential
Kakihana et al., J Thorac Oncol 2009 (Carcinoma, Non-Small-Cell Lung...) : In contrast to H460 and H661, H157 cells were resistant to E-cadherin up-regulation by HDAC inhibitors
Li et al., J Biol Chem 2010 (Prostatic Neoplasms) : In further analysis we find that retinoblastoma binding protein 7 is a transcriptional activator of E-cadherin and that its intercellular transfer leads to decreased gene expression of downstream targets such as N-cadherin and vimentin
Huang et al., Mol Pharmacol 2011 : This finding, together with the HDAC inhibitor induced up-regulation of KLF4 and E-cadherin , suggests that HDAC inhibitors could activate the expression of these genes through changes in histone methylation status ... This finding, together with the HDAC inhibitor induced up-regulation of KLF4 and E-cadherin , suggests that HDAC inhibitors could activate the expression of these genes through changes in histone methylation status
Yi et al., Chemotherapy 2012 (Carcinoma...) : DNMT inhibitors and HDAC inhibitors regulate E-cadherin and Bcl-2 expression in endometrial carcinoma in vitro and in vivo
Nagathihalli et al., Mol Cancer Ther 2012 (Carcinoma, Non-Small-Cell Lung...) : Smoking induces epithelial-to-mesenchymal transition in non-small cell lung cancer through HDAC mediated downregulation of E-cadherin
Iordache et al., International journal of molecular sciences 2012 : RT-PCR assay showed that HDAC inhibitors down-regulated the expression of endothelial genes such as VE-cadherin , CD133, CXCR4 and Tie-2