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IL13 — SOAT1
Text-mined interactions from Literome
Guiter et al., Blood 2004
(Lymphoma, B-Cell...) :
IL-4 and IL-13 transcripts were absent in PMBL cell lines and expressed at low levels in tumors, indicating that, contrary to classical Hodgkin lymphoma ( cHL ),
STAT6 activation is not
due to an autocrine
IL-4/IL-13 secretion
Bian et al., Proc Natl Acad Sci U S A 2005
:
We now demonstrate that ( i ) an IL-4 receptor alpha-subunit ( IL-4Ralpha )
/Stat6 dependent but T cell independent pathway is the key for the nematode induced host NOS-2 inhibition ; ( ii ) endogenous IL-4 and
IL-13 , the only known IL-4Ralpha ligands, are not
required for activating the pathway ; and ( iii ) treatment of RAW264.7 cells with parasite cultured medium inhibits NOS-2 expression but not cyclooxygenase 2 expression
Chiba et al., J Smooth Muscle Res 2009
(Asthma...) :
In the present study, the
effects of
interleukin-13 (IL-13) and IL-4 on activation of these
STAT molecules were examined in cultured human bronchial smooth muscle cells ( hBSMCs )
Goenka et al., Immunol Res 2011
:
Among the seven STAT proteins,
STAT6 is
activated by IL-4 and
IL-13 and plays a predominant role in the immune system
Russell et al., Islets 2013
:
Differential
effects of
interleukin-13 and interleukin-6 on
Jak/STAT signaling and cell viability in pancreatic ß-cells
Seidel et al., Proc Natl Acad Sci U S A 1995
(Carcinoma, Hepatocellular...) :
We have systematically examined the effects of the spacing between the TT and AA core half sites on the binding of the
STAT complexes
activated by IFN-gamma,
interleukin (IL) 6, granulocyte-macrophage colony stimulating factor, and IL-4
Murata et al., J Immunol 1996
(Colonic Neoplasms) :
Both
IL-13 and IL-4
induced phosphorylation of IL-4
STAT ( STAT6 ) but not STAT1, STAT3, or STAT5